rs146907080

This variant is located in the SPECC1L gene.

ClinVar annotation

Likely Benign☆☆☆
2 submitters1 publication

not provided; SPECC1L-related disorder

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Research that mentions this SNP (1)

MCS9.7 enhancer activity is highly, but not completely, associated with expression of Irf6 and p63
FunctionalWalid D. Fakhouri et al.(2012)· Developmental Dynamics

This dissertation characterizes SPECC1L function in palatogenesis using mouse models and identifies SPECC1L variants in human patients with orofacial clefts. The authors demonstrate that SPECC1L is required for palate elevation and that the T397P point mutation identified in Opitz G/BBB syndrome patients results in completely penetrant cleft palate with omphalocele phenotypes in mouse embryos. SPECC1L acts downstream of IRF6 in the palatal epithelium and is required for mesenchymal cell migration, with defects rescuable by PI3K-AKT pathway activation.

Traits studied:Cleft palateOpitz G/BBB syndromeOrofacial cleftTeebi hypertelorism syndrome

About SPECC1L

This gene encodes a coiled-coil domain containing protein. The encoded protein may play a critical role in actin-cytoskeletal reorganization during facial morphogenesis. Mutations in this gene are a cause of oblique facial clefting-1. Alternatively spliced transcript variants encoding multiple isoforms have been observed for this gene. A read-through transcript composed of SPECC1L (sperm antigen with calponin homology and coiled-coil domains 1-like) and the downstream ADORA2A (adenosine A2a receptor) gene sequence has been identified, but it is thought to be non-coding. [provided by RefSeq, Jun 2013]

View all SPECC1L variants →

Gene information from NCBI Gene. Variant classifications from ClinVar.

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