rs4947534

This is a 3 prime utr variant variant in the PSPH gene.

GWAS Catalog Trait Associations (3)

Genome-wide significant associations (p < 5×10⁻⁸) from the NHGRI-EBI GWAS Catalog.

serine measurement

Allele C
OR 17.31
p 4.0e-67
N 30,955
Large GWAS
European
Shin SY et al. An atlas of genetic influences on human blood metabolites. Nature Genetics 46(6):543-550 (2014)
Allele C
OR
β 0.018
p 2.0e-14
N 7,796
Large GWAS
European
Allele C
OR
β 0.041
p 4.0e-18
N 7,478
Large GWAS
European

glycine measurement

Wittemans LBL et al. Assessing the causal association of glycine with risk of cardio-metabolic diseases. Nature Communications 10(1):1060 (2019)
Allele C
OR 0.07
p 7.0e-34
N 80,003
Large GWAS
European
Allele C
OR 12.13
p 7.0e-34
N 80,003
Large GWAS
European

serum alanine aminotransferase amount

Allele T
OR 0.02
p 5.0e-29
N 928,679
Large GWAS
multi-ancestry
Sakaue S et al. A cross-population atlas of genetic associations for 220 human phenotypes. Nature Genetics 53(10):1415-1424 (2021)
Allele T
OR 0.02
p 2.0e-13
N 494,681
Large GWAS
multi-ancestry

ClinVar annotation

Benign☆☆☆
2 submitters1 publication

Deficiency of phosphoserine phosphatase (PSPHD)

View on ClinVar →

About PSPH

The protein encoded by this gene belongs to a subfamily of the phosphotransferases. This encoded enzyme is responsible for the third and last step in L-serine formation. It catalyzes magnesium-dependent hydrolysis of L-phosphoserine and is also involved in an exchange reaction between L-serine and L-phosphoserine. Deficiency of this protein is thought to be linked to Williams syndrome. [provided by RefSeq, Jul 2008]

View all PSPH variants →

Gene information from NCBI Gene. Variant classifications from ClinVar.

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