rs719149

This is a regulatory region variant variant in the TNFAIP3 gene.

ClinVar annotation

Benign☆☆☆
2 submitters1 publication

not specified

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Research that mentions this SNP (2)

Association of single‐nucleotide polymorphisms in CCR6, TAGAP, and TNFAIP3 with rheumatoid arthritis in African Americans
AssociationN=1,179Perkins EA et al.(2012)· Arthritis & Rheumatism

This case-control study of 446 African-American RA patients and 733 controls identified three SNPs significantly associated with rheumatoid arthritis: TNFAIP3 rs719149 (A allele OR 1.22, p=0.02), TAGAP rs1738074 (G allele OR 0.75, p=0.0012), and TAGAP rs4709267 (G allele OR 0.74, p=0.004). Conditional analyses suggest the two TAGAP SNPs have independent effects despite weak linkage disequilibrium (R²=0.034).

Traits studied:Rheumatoid arthritis
Association of TNFAIP3 polymorphism with rheumatic heart disease in Chinese Han population
AssociationN=717Rong Hua et al.(2009)· Immunogenetics

A pair-matched case-control study (239 cases, 478 controls) in the Chinese Han population identified rs582757 in the TNFAIP3 gene as associated with reduced risk of rheumatic heart disease (RHD). The minor C allele showed a per-allele odds ratio of 0.57 (95% CI 0.42-0.78, p=0.000) in the additive model and a 0.54-fold reduced risk under the dominant model. No significant associations were found for SNPs in the TRAF1 or C5 genes.

Traits studied:Rheumatic heart disease

About TNFAIP3

This gene was identified as a gene whose expression is rapidly induced by the tumor necrosis factor (TNF). The protein encoded by this gene is a zinc finger protein and ubiqitin-editing enzyme, and has been shown to inhibit NF-kappa B activation as well as TNF-mediated apoptosis. The encoded protein, which has both ubiquitin ligase and deubiquitinase activities, is involved in the cytokine-mediated immune and inflammatory responses. Several transcript variants encoding the same protein have been found for this gene. [provided by RefSeq, Jul 2012]

View all TNFAIP3 variants →

Gene information from NCBI Gene. Variant classifications from ClinVar.

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